Learned helplessness
Learned helplessness is the behavior exhibited by a subject after enduring repeated aversive stimuli beyond their control. It was initially thought to be caused by the subject's acceptance of their powerlessness, by way of their discontinuing attempts to escape or avoid the aversive stimulus, even when such alternatives are unambiguously presented. Upon exhibiting such behavior, the subject was said to have acquired learned helplessness.[1][2] Over the past few decades, neuroscience has provided insight into learned helplessness and shown that the original theory had it backward: the brain's default state is to assume that control is not present, and the presence of "helplessness" is what is learned first. However, it is unlearned when a subject is faced with prolonged aversive stimulation.[3]
In humans, learned helplessness is related to the concept of self-efficacy; the individual's belief in their innate ability to achieve goals. Learned helplessness theory is the view that clinical depression and related mental illnesses may result from a real or perceived absence of control over the outcome of a situation.[4]
Research has found that a human's reaction to feeling a lack of control differs both between individuals and between situations, i.e. learned helplessness sometimes remains specific to one situation but at other times generalizes across situations.[7][10][11] Such variations are not explained by the original theory of learned helplessness, and an influential view is that such variations depend on an individual's attributional or explanatory style.[12] According to this view, how someone interprets or explains adverse events affects their likelihood of acquiring learned helplessness and subsequent depression.[13] For example, people with pessimistic explanatory style tend to see negative events as permanent ("it will never change"), personal ("it's my fault"), and pervasive ("I can't do anything correctly"), and are likely to suffer from learned helplessness and depression.[14]
In 1978, Lyn Yvonne Abramson, Seligman, Paul and John D. Teasdale reformulated Seligman's & Paul's work, using attribution theory. They proposed that people differed in how they classified negative experiences on three scales, from internal to external, stable to unstable, and from global to specific. They believed that people who were more likely to attribute negative events to internal, stable, and global causes were more likely to become depressed than those who attributed things to causes at the other ends of the scales.[13]
Bernard Weiner proposed a detailed account of the attributional approach to learned helplessness in 1986. His attribution theory includes the dimensions of globality/specificity, stability/instability, and internality/externality:[15]
Neurobiological perspective[edit]
Research has shown that increased 5-HT (serotonin) activity in the dorsal raphe nucleus plays a critical role in learned helplessness. Other key brain regions that are involved with the expression of helpless behavior include the basolateral amygdala, central nucleus of the amygdala and
bed nucleus of the stria terminalis.[16] Activity in medial prefrontal cortex, dorsal hippocampus, septum and hypothalamus has also been observed during states of helplessness.
In the article, "Exercise, Learned Helplessness, and the Stress-Resistant Brain", Benjamin N. Greenwood and Monika Fleshner discuss how exercise might prevent stress-related disorders such as anxiety and depression. They show evidence that running wheel exercise prevents learned helplessness behaviors in rats.[17] They suggest that the amount of exercise may not be as important as simply exercising at all. The article also discusses the neurocircuitry of learned helplessness, the role of serotonin (or 5-HT), and the exercise-associated neural adaptations that may contribute to the stress-resistant brain. However, the authors finally conclude that "The underlying neurobiological mechanisms of this effect, however, remain unknown. Identifying the mechanisms by which exercise prevents learned helplessness could shed light on the complex neurobiology of depression and anxiety and potentially lead to novel strategies for the prevention of stress-related mood disorders".[18]
In developmental psychology the order of various stages of neurobiological development is important. From this perspective there are two different kinds of "helplessness" that appear at different stages of development. In early development, the infant is naturally helpless and must learn "helpfulness" toward mature neurophysiology. The "helplessness" that appears after maturation is what is properly termed "learned helplessness", although some researchers conflate this infantile form of "helplessness" with the pathological, adult, form.[19]
Extensions[edit]
Cognitive scientist and usability engineer Donald Norman used learned helplessness to explain why people blame themselves when they have a difficult time using simple objects in their environment.[47]
The U.S. sociologist Harrison White has suggested in his book Identity and Control that the notion of learned helplessness can be extended beyond psychology into the realm of social action. When a culture or political identity fails to achieve desired goals, perceptions of collective ability suffer.
Emergence in the political atmosphere[edit]
In a political setting, learned helplessness is involved when a voter votes for a candidate and that candidate does not win.[48] If this happens over time, it can lead to learned helplessness. When this does occur, it can often lead to having fewer voters in the future. However, Wollman & Stouder (1991) found that there was not a significant finding between situation-specific efficacy and predictive behavior of voting.[49]
Emergence under torture[edit]
Studies on learned helplessness served as the basis for developing American torture methods. In CIA interrogation manuals, learned helplessness is characterized as "apathy" which may result from prolonged use of coercive techniques which result in a "debility-dependency-dread" state in the subject, "If the debility-dependency-dread state is unduly prolonged, however, the arrestee may sink into a defensive apathy from which it is hard to arouse him."[50][51]