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Transcription (biology)

Transcription is the process of copying a segment of DNA into RNA. The segments of DNA transcribed into RNA molecules that can encode proteins produce messenger RNA (mRNA). Other segments of DNA are transcribed into RNA molecules called non-coding RNAs (ncRNAs).

This article is about transcription in biology. For other uses, see Transcription.

Both DNA and RNA are nucleic acids, which use base pairs of nucleotides as a complementary language. During transcription, a DNA sequence is read by an RNA polymerase, which produces a complementary, antiparallel RNA strand called a primary transcript.


In virology, the term transcription may also be used when referring to mRNA synthesis from an RNA molecule (i.e., equivalent to RNA replication). For instance, the genome of a negative-sense single-stranded RNA (ssRNA -) virus may be a template for a positive-sense single-stranded RNA (ssRNA +). This is because the positive-sense strand contains the sequence information needed to translate the viral proteins needed for viral replication. This process is catalyzed by a viral RNA replicase.[1]

Background[edit]

A DNA transcription unit encoding for a protein may contain both a coding sequence, which will be translated into the protein, and regulatory sequences, which direct and regulate the synthesis of that protein. The regulatory sequence before (upstream from) the coding sequence is called the five prime untranslated regions (5'UTR); the sequence after (downstream from) the coding sequence is called the three prime untranslated regions (3'UTR).[2]


As opposed to DNA replication, transcription results in an RNA complement that includes the nucleotide uracil (U) in all instances where thymine (T) would have occurred in a DNA complement.[3]


Only one of the two DNA strands serves as a template for transcription. The antisense strand of DNA is read by RNA polymerase from the 3' end to the 5' end during transcription (3' → 5'). The complementary RNA is created in the opposite direction, in the 5' → 3' direction, matching the sequence of the sense strand except switching uracil for thymine. This directionality is because RNA polymerase can only add nucleotides to the 3' end of the growing mRNA chain. This use of only the 3' → 5' DNA strand eliminates the need for the Okazaki fragments that are seen in DNA replication.[2] This also removes the need for an RNA primer to initiate RNA synthesis, as is the case in DNA replication.


The non-template (sense) strand of DNA is called the coding strand, because its sequence is the same as the newly created RNA transcript (except for the substitution of uracil for thymine). This is the strand that is used by convention when presenting a DNA sequence.[4]


Transcription has some proofreading mechanisms, but they are fewer and less effective than the controls for copying DNA. As a result, transcription has a lower copying fidelity than DNA replication.[5]

Inhibitors[edit]

Transcription inhibitors can be used as antibiotics against, for example, pathogenic bacteria (antibacterials) and fungi (antifungals). An example of such an antibacterial is rifampicin, which inhibits bacterial transcription of DNA into mRNA by inhibiting DNA-dependent RNA polymerase by binding its beta-subunit, while 8-hydroxyquinoline is an antifungal transcription inhibitor.[54] The effects of histone methylation may also work to inhibit the action of transcription. Potent, bioactive natural products like triptolide that inhibit mammalian transcription via inhibition of the XPB subunit of the general transcription factor TFIIH has been recently reported as a glucose conjugate for targeting hypoxic cancer cells with increased glucose transporter production.[55]

History[edit]

A molecule that allows the genetic material to be realized as a protein was first hypothesized by François Jacob and Jacques Monod. Severo Ochoa won a Nobel Prize in Physiology or Medicine in 1959 for developing a process for synthesizing RNA in vitro with polynucleotide phosphorylase, which was useful for cracking the genetic code. RNA synthesis by RNA polymerase was established in vitro by several laboratories by 1965; however, the RNA synthesized by these enzymes had properties that suggested the existence of an additional factor needed to terminate transcription correctly.


Roger D. Kornberg won the 2006 Nobel Prize in Chemistry "for his studies of the molecular basis of eukaryotic transcription".[61]

transcription assay: measures promoter strength

G-Less Cassette

assay: identifies transcription start sites (TSS)

Run-off transcription

assay: measures the relative abundance of newly formed transcripts

Nuclear run-on

: measures single-stranded DNA generated by RNA polymerases; can work with 1,000 cells.[62]

KAS-seq

and ChIP-Chip of RNAP: detect active transcription sites

RNase protection assay

: measures the absolute abundance of total or nuclear RNA levels, which may however differ from transcription rates

RT-PCR

: measures the relative abundance of the global total or nuclear RNA levels; however, these may differ from transcription rates

DNA microarrays

: detects the presence of a transcript

In situ hybridization

: by incorporating RNA stem loops, such as MS2, into a gene, these become incorporated into newly synthesized RNA. The stem loops can then be detected using a fusion of GFP and the MS2 coat protein, which has a high affinity, sequence-specific interaction with the MS2 stem loops. The recruitment of GFP to the site of transcription is visualized as a single fluorescent spot. This new approach has revealed that transcription occurs in discontinuous bursts, or pulses (see Transcriptional bursting). With the notable exception of in situ techniques, most other methods provide cell population averages, and are not capable of detecting this fundamental property of genes.[63]

MS2 tagging

: the traditional method, and until the advent of RNA-Seq, the most quantitative

Northern blot

: applies next-generation sequencing techniques to sequence whole transcriptomes, which allows the measurement of relative abundance of RNA, as well as the detection of additional variations such as fusion genes, post-transcriptional edits and novel splice sites

RNA-Seq

: amplifies and reads partial transcriptomes from isolated cells, allowing for detailed analyses of RNA in tissues, embryos, and cancers

Single cell RNA-Seq

Transcription can be measured and detected in a variety of ways:

Life

Cell (biology)

Cell division

DBTSS

gene

gene regulation

gene expression

Epigenetics

Genome

Gene regulation

Long non-coding RNA

Missense mRNA

– process of removing introns from precursor messenger RNA (pre-mRNA) to make messenger RNA (mRNA)

Splicing

Transcriptomics

Translation (biology)

Archived 2011-07-22 at the Wayback Machine From Center for Models of Life Archived 2011-08-09 at the Wayback Machine at the Niels Bohr Institute.

Interactive Java simulation of transcription initiation.

Archived 2011-08-26 at the Wayback Machine From Center for Models of Life Archived 2011-08-09 at the Wayback Machine at the Niels Bohr Institute.

Interactive Java simulation of transcription interference—a game of promoter dominance in bacterial virus.

Archived 2021-04-14 at the Wayback Machine

Virtual Cell Animation Collection, Introducing Transcription